07-11-2022-1115 - 7119/20-21,22-23
Monday, July 11, 2022
07-11-2022-0809 - capillary leak syndrome Cyclophosphamide granulomatosis polyangitis vasculitides ANCA anti neutrophil cytoplasmic antibody antibodies agglomerates tissue generators subtis generator deviation sub repair detachment giant large cell turmor of bonules autoantibodies divergence of process
Capillary leak syndrome is characterized by the escape of blood plasma through capillary walls, from the blood circulatory system to surrounding tissues, muscle compartments, organs or body cavities. It is a phenomenon most commonly witnessed in sepsis, and less frequently in autoimmune diseases, differentiation syndrome, engraftment syndrome, hemophagocytic lymphohistiocytosis, the ovarian hyperstimulation syndrome, viral hemorrhagic fevers, and snakebite and ricin poisoning.[1] Pharmaceuticals, including the chemotherapy medications gemcitabine and tagraxofusp, as well as certain interleukins and monoclonal antibodies, can also cause capillary leaks.[1] These conditions and factors are sources of secondary capillary leak syndrome.
https://en.wikipedia.org/wiki/Capillary_leak_syndrome
Cyclophosphamide (CP), also known as cytophosphane among other names,[3] is a medication used as chemotherapyand to suppress the immune system.[4] As chemotherapy it is used to treat lymphoma, multiple myeloma, leukemia, ovarian cancer, breast cancer, small cell lung cancer, neuroblastoma, and sarcoma.[4] As an immune suppressor it is used in nephrotic syndrome, granulomatosis with polyangiitis, and following organ transplant, among other conditions.[4][5]It is taken by mouth or injection into a vein.[4]
Most people develop side effects.[4] Common side effects include low white blood cell counts, loss of appetite, vomiting, hair loss, and bleeding from the bladder.[4] Other severe side effects include an increased future risk of cancer, infertility, allergic reactions, and pulmonary fibrosis.[4] Cyclophosphamide is in the alkylating agent and nitrogen mustard family of medications.[4] It is believed to work by interfering with the duplication of DNA and the creation of RNA.[4]
Cyclophosphamide was approved for medical use in the United States in 1959.[4] It is on the World Health Organization's List of Essential Medicines.[6]
https://en.wikipedia.org/wiki/Cyclophosphamide
Granulomatosis with polyangiitis (GPA), previously known as Wegener's granulomatosis (WG),[1][2][3][4][5] is a rare long-term systemic disorder that involves the formation of granulomas and inflammation of blood vessels(vasculitis). It is a form of vasculitis that affects small- and medium-size vessels in many organs but most commonly affects the upper respiratory tract, lungs and kidneys.[6] The signs and symptoms of GPA are highly varied and reflect which organs are supplied by the affected blood vessels. Typical signs and symptoms include nosebleeds, stuffy nose and crustiness of nasal secretions, and inflammation of the uveal layer of the eye.[3]Damage to the heart, lungs and kidneys can be fatal.
The cause of GPA is unknown. Genetics have been found to play a role in GPA though the risk of inheritance appears to be low.[7]
GPA treatment depends on the severity of the disease.[8] Severe disease is typically treated with a combination of immunosuppressive medications such as rituximab or cyclophosphamide and high-dose corticosteroids to control the symptoms of the disease and azathioprine, methotrexate, or rituximab to keep the disease under control.[1][7][8]Plasma exchange is also used in severe cases with damage to the lungs, kidneys, or intestines.[9]
The number of new cases of GPA each year is estimated to be 2.1–14.4 new cases per million people in Europe.[3]GPA is rare in Japanese and African-American populations but occurs more often in people of Northern European descent.[7] GPA is estimated to affect 3 cases per 100,000 people in the United States and equally affects men and women.[10]
https://en.wikipedia.org/wiki/Granulomatosis_with_polyangiitis
ANCAs are associated with small vessel vasculitides including granulomatosis with polyangiitis, microscopic polyangiitis, primary pauci-immune necrotizing crescentic glomerulonephritis (a type of renal-limited microscopic polyangiitis), eosinophilic granulomatosis with polyangiitis and drug induced vasculitides. PR3 directed c-ANCA is present in 80-90% of granulomatosis with polyangiitis, 20-40% of microscopic polyangiitis, 20-40% of pauci-immune crescentic glomerulonephritis and 35% of eosinophilic granulomatosis with polyangiitis. c-ANCA (atypical) is present in 80% of cystic fibrosis (with BPI as the target antigen) and also in inflammatory bowel disease, primary sclerosing cholangitis and rheumatoid arthritis (with antibodies to multiple antigenic targets). p-ANCA with MPO specificity is found in 50% of microscopic polyangiitis, 50% of primary pauci-immune necrotizing crescentic glomerulonephritis and 35% of eosinophilic granulomatosis with polyangiitis. p-ANCA with specificity to other antigens are associated with inflammatory bowel disease, rheumatoid arthritis, drug-induced vasculitis, autoimmune liver disease, drug induced syndromes and parasitic infections. Atypical ANCA is associated with drug-induced systemic vasculitis, inflammatory bowel disease and rheumatoid arthritis.[2][4] The ANCA‐positive rate is much higher in patients with type 1 diabetes mellitus than in healthy individuals.[5]
Levamisole, which is a common adulterant of cocaine, can cause an ANCA positive vasculitis.[6]
The presence or absence of ANCA cannot indicate presence or absence of disease and results are correlated with clinical features. The association of ANCA and disease activity remains controversial; however, the reappearance of ANCA after treatment can indicate a relapse.[7][8]
https://en.wikipedia.org/wiki/Anti-neutrophil_cytoplasmic_antibody#Disease_associations
https://en.wikipedia.org/wiki/Capillary_leak_syndrome
Cyclophosphamide (CP), also known as cytophosphane among other names,[3] is a medication used as chemotherapyand to suppress the immune system.[4] As chemotherapy it is used to treat lymphoma, multiple myeloma, leukemia, ovarian cancer, breast cancer, small cell lung cancer, neuroblastoma, and sarcoma.[4] As an immune suppressor it is used in nephrotic syndrome, granulomatosis with polyangiitis, and following organ transplant, among other conditions.[4][5]It is taken by mouth or injection into a vein.[4]
Most people develop side effects.[4] Common side effects include low white blood cell counts, loss of appetite, vomiting, hair loss, and bleeding from the bladder.[4] Other severe side effects include an increased future risk of cancer, infertility, allergic reactions, and pulmonary fibrosis.[4] Cyclophosphamide is in the alkylating agent and nitrogen mustard family of medications.[4] It is believed to work by interfering with the duplication of DNA and the creation of RNA.[4]
Cyclophosphamide was approved for medical use in the United States in 1959.[4] It is on the World Health Organization's List of Essential Medicines.[6]
https://en.wikipedia.org/wiki/Cyclophosphamide
Granulomatosis with polyangiitis (GPA), previously known as Wegener's granulomatosis (WG),[1][2][3][4][5] is a rare long-term systemic disorder that involves the formation of granulomas and inflammation of blood vessels(vasculitis). It is a form of vasculitis that affects small- and medium-size vessels in many organs but most commonly affects the upper respiratory tract, lungs and kidneys.[6] The signs and symptoms of GPA are highly varied and reflect which organs are supplied by the affected blood vessels. Typical signs and symptoms include nosebleeds, stuffy nose and crustiness of nasal secretions, and inflammation of the uveal layer of the eye.[3]Damage to the heart, lungs and kidneys can be fatal.
The cause of GPA is unknown. Genetics have been found to play a role in GPA though the risk of inheritance appears to be low.[7]
GPA treatment depends on the severity of the disease.[8] Severe disease is typically treated with a combination of immunosuppressive medications such as rituximab or cyclophosphamide and high-dose corticosteroids to control the symptoms of the disease and azathioprine, methotrexate, or rituximab to keep the disease under control.[1][7][8]Plasma exchange is also used in severe cases with damage to the lungs, kidneys, or intestines.[9]
The number of new cases of GPA each year is estimated to be 2.1–14.4 new cases per million people in Europe.[3]GPA is rare in Japanese and African-American populations but occurs more often in people of Northern European descent.[7] GPA is estimated to affect 3 cases per 100,000 people in the United States and equally affects men and women.[10]
https://en.wikipedia.org/wiki/Granulomatosis_with_polyangiitis
ANCAs are associated with small vessel vasculitides including granulomatosis with polyangiitis, microscopic polyangiitis, primary pauci-immune necrotizing crescentic glomerulonephritis (a type of renal-limited microscopic polyangiitis), eosinophilic granulomatosis with polyangiitis and drug induced vasculitides. PR3 directed c-ANCA is present in 80-90% of granulomatosis with polyangiitis, 20-40% of microscopic polyangiitis, 20-40% of pauci-immune crescentic glomerulonephritis and 35% of eosinophilic granulomatosis with polyangiitis. c-ANCA (atypical) is present in 80% of cystic fibrosis (with BPI as the target antigen) and also in inflammatory bowel disease, primary sclerosing cholangitis and rheumatoid arthritis (with antibodies to multiple antigenic targets). p-ANCA with MPO specificity is found in 50% of microscopic polyangiitis, 50% of primary pauci-immune necrotizing crescentic glomerulonephritis and 35% of eosinophilic granulomatosis with polyangiitis. p-ANCA with specificity to other antigens are associated with inflammatory bowel disease, rheumatoid arthritis, drug-induced vasculitis, autoimmune liver disease, drug induced syndromes and parasitic infections. Atypical ANCA is associated with drug-induced systemic vasculitis, inflammatory bowel disease and rheumatoid arthritis.[2][4] The ANCA‐positive rate is much higher in patients with type 1 diabetes mellitus than in healthy individuals.[5]
Levamisole, which is a common adulterant of cocaine, can cause an ANCA positive vasculitis.[6]
The presence or absence of ANCA cannot indicate presence or absence of disease and results are correlated with clinical features. The association of ANCA and disease activity remains controversial; however, the reappearance of ANCA after treatment can indicate a relapse.[7][8]
https://en.wikipedia.org/wiki/Anti-neutrophil_cytoplasmic_antibody#Disease_associations
Autoantibodies
Arteriosclerosis obliterans is an occlusive arterial disease most prominently affecting the abdominal aorta and the small- and medium-sized arteries of the lower extremities, which may lead to absent dorsalis pedis, posterior tibial, and/or popliteal artery pulses.[1]: 842
It is characterized by fibrosis of the tunica intima and calcification of the tunica media.
https://en.wikipedia.org/wiki/Arteriosclerosis_obliterans
Superficial thrombophlebitis is a thrombosis and inflammation of superficial veins which presents as a painful indurationwith erythema, often in a linear or branching configuration forming cords.[2]: 826–7 [3]
Superficial thrombophlebitis is due to inflammation and/or thrombosis, and less commonly infection of the vein. It is generally a benign, self-limited disorder, however, it can be complicated by deep vein thrombosis (DVT) and even pulmonary embolism (PE)[4] Migratory superficial thrombophlebitis is known as Trousseau's syndrome.[5]
https://en.wikipedia.org/wiki/Superficial_thrombophlebitis
https://www.cancer.gov/publications/dictionaries/cancer-terms/def/capillary
Angiostrongyliasis is an infection by a roundworm of the Angiostrongylus type. Symptoms may vary from none, to mild, to meningitis.[1]
Infection with Angiostrongylus cantonensis (rat lungworm) can occur after ingestion of raw or undercooked snails or slugs, and less likely unwashed fruits and vegetables.
In humans, A. cantonensis is the most common cause of eosinophilic meningitis or meningoencephalitis.[2] Frequently the infection will resolve without treatment or serious consequences, but in cases with a heavy load of parasites the infection can be so severe it can cause permanent damage to the central nervous system or death.[3]
https://en.wikipedia.org/wiki/Angiostrongyliasis
Meningoencephalitis (/mɪˌnɪŋɡoʊɛnˌsɛfəˈlaɪtɪs, -ˌnɪndʒoʊ-, -ən-, -ˌkɛ-/;[3][4] from Greek μῆνιγξ meninx, "membrane", ἐγκέφαλος, enképhalos "brain", and the medical suffix -itis, "inflammation"), also known as herpes meningoencephalitis, is a medical condition that simultaneously resembles both meningitis, which is an infectionor inflammation of the meninges, and encephalitis, which is an infection or inflammation of the brain.
Signs and symptoms[edit]
Signs of meningeoncephalitis include unusual behavior, personality changes, and thinking problems.[5][6]
Symptoms may include headache, fever, pain in neck movement, light sensitivity, and seizure.[7]
Causes[edit]
Causative organisms include protozoans, viral and bacterial pathogens.[citation needed]
Specific types include:
Bacterial[edit]
Veterinarians have observed meningoencephalitis in animals infected with listeriosis, caused by the pathogenic bacteria L. monocytogenes. Meningitis and encephalitis already present in the brain or spinal cord of an animal may form simultaneously into meningeoencephalitis.[8] The bacterium commonly targets the sensitive structures of the brain stem. L. monocytogenes meningoencephalitis has been documented to significantly increase the number of cytokines, such as IL-1β, IL-12, IL-15, leading to toxic effects on the brain.[9]
Meningoencephalitis may be one of the severe complications of diseases originating from several Rickettsia species, such as Rickettsia rickettsii (agent of Rocky Mountain spotted fever (RMSF)), Rickettsia conorii, Rickettsia prowazekii (agent of epidemic louse-borne typhus), and Rickettsia africae. It can cause impairments to the cranial nerves, paralysis to the eyes, and sudden hearing loss.[10][11] Meningoencephalitis is a rare, late-stage manifestation of tick-borne ricksettial diseases, such as RMSF and Human monocytotropic ehrlichiosis (HME), caused by Ehrlichia chaffeensis (a species of rickettsiales bacteria).[12]
Other bacteria that can cause it are Mycoplasma pneumoniae, Tuberculosis, Borrelia (Lyme disease) and Leptospirosis.
Viral[edit]
- Tick-borne encephalitis
- West Nile virus
- Measles
- Epstein-Barr virus
- Varicella-zoster virus
- Enterovirus
- Herpes simplex virus type 1
- Herpes simplex virus type 2
- Rabies virus
- Adenovirus, although meningoencephalitis is almost solely seen in heavily immunocompromised patients.[13]
- Mumps, a relatively common cause of meningoencephalitis. However, most cases are mild, and mumps meningoencephalitis generally does not result in death or neurologic sequelae.[14]
- HIV, a very small number of individuals exhibit meningoencephalitis at the primary stage of infection.[15][16]
Autoimmune[edit]
- Antibodies targeting amyloid beta peptide proteins which have been used during research on Alzheimer's disease.[17]
- Anti-N-methyl-D-aspartate (anti-NMDA) receptor antibodies, which are also associated with seizures and a movement disorder, and related to Anti-NMDA receptor encephalitis.
- NAIM or "Nonvasculitic autoimmune inflammatory meningoencephalitis" (NAIM).[18] They can be divided into GFAP- and GFAP+ cases. The second is related to the Autoimmune GFAP Astrocytopathy.
Protozoal[edit]
Animal[edit]
This nematode is an exceptionally rare cause of meningoencephalitis.[19]
Other/multiple[edit]
Other causes include granulomatous meningoencephalitis and vasculitis. The fungus, Cryptococcus neoformans, can be symptomatically manifested within the CNS as meningoencephalitis with hydrocephalus being a very characteristic finding due to the unique thick polysaccharide capsule of the organism.[citation needed]
https://en.wikipedia.org/wiki/Meningoencephalitis
https://en.wikipedia.org/wiki/Cryptococcus_neoformans
07-10-2022-1058 - art etc..
LUDAVERSE
JUMP IN POOLS AND MAKE A BIG SPLASH
ACT A FOOL
B R RIGHT
MINI GREG (MEDICAL CHEIF)
BOBROCHIMARYU & KIMMYMARYU
OROCHIMARU & KIMIMARO
DR. BETTEY RESEMBLANCE MEN
HARRY POTTER & THE SORCERS STONE... BOY SCIENTIST
BLAS TO THE PAST
LORD OF THE RINGS (GIVE ME THE RING FRODO..GOLEMN & FRODO @.CHRISTOPHER WALSH ROMANINY V. NIKI ANTON BETTEY)
GRANDMA RUBY MAY, RABYS WEB, GANMA TUBY, GRAN RUBES, LA RUBY MAY, ETC..
MR PERRY & LE FREUS CHERIE
YEAH LIKE THEY JUST GONNA FIGHT THE BREAKER
SNOW WHITE
GERMAN PEACH
ASSPHRODIYTE
VASILIKI BLACK CURLY HEAD HAIR WHITE SKIN CAUCASIAN MIDDLE EAST GREEK VAR EUROPE UNK
SPACE ALIEN
FRACKLE PUFF
N-SPACE FIGHTER
DARTH VADER V. LUKE
NUKE SKYWALKER
NAKED GUN
MAN WHO KNEW TOO LITTLE
AGENT MULLER
BASE
BLACK BLACK
SCOTT PETERSEN DOUBLE VAR SPECIAL FORCES LORD
PEATTY WADE
XLIF VIV BIRTHRIGHT
PEETER PIENER
DRESS LIKE YOUR TEACHER
CRUSADERS AND SNOW WHITE
NO REFLECTION AND THE VIRGIN SUICIDES PEOPLE CHRISTIANS
MARIE ANTOINETTE LAVOSIER
SS WAFFLE BAT
WITCHCRAFT BROTHER
RACE BAG
NORA FRIES, LAST UNICORN, KIMIMARO
ANTELOPINI, FURRCHINI
Leptospirochete, spirulina, spirochinis, midgie tape, leish, helstr, asscaries, fruke, hektor strip, bitsh, hook, thread, thrip, tape, heart see, c worm, lil chubby, lash, dros, name the worm, nemi, tod, hel, can, tub, strep, skar, puck, bundle pack, fiber bundle, conductor, insect, popcon, mini leperchaun, ghas, gan, black worm, worm scar, xlif worm, white worm, gris, scrab, ulka, prot, boid, ame, mac, phag, co pin, mal, vir, auto, nuc, agglo, lys, plat, sponge, fung, nag, flow mod, drain stopper, cap screen, occluder, cas, cat, denny, root, can, mole, hole, flit, lar, prio, frag, spinode, bor, flat, round, long duck dong, slim jim, rolly, striated, formed, semi sebulated lifeform, pus reform, incarnation, mag, sol gel semi sol semi gel vars, party, peat gang, liver rock, cyscercryst, crhist, cap, jun, mat, blacks, deep drill, organ starter, hemi, retro, thfrom, swamp, dampner, solva, seeper, cap inf vir, filarial, clot pert pyo, fluzie, marcerens, rick, sia, anna, typhus, therus, dysts, chole ria, dia, drac, gran, pole, hygrags, hygroscop, cormyco, ostnec, avas, ossi, myel, liti, pyre, gen, deta, game time, caudo phi ikz, syntax, skele, synaps, sinners, sin, connexor, tubulas, filiment, struc brac, anchor, hole fiber, schisto, mansoni, carb chond, chondrocyte, cor col cordite glycol, denditius reticulus (dentition reticulum), vulum, tabes, glut, granuloma, dry proc des met foam fly ash blower acid salt pres pre spinor plate thin filmdep gas line part array grad mag fold spin thread weave melt smelt recon mat acc grav accrete jet str imp exp homog centrifuge spiral circ cen ac vert alter nat vac dry flash etc., shear drag hydrag prop salt generator, shear stress strain rotary, mirror, gas lien lasar, pressure reactor, mixer, high shear mixer, homogenizer, calibration measure environment system surround, uranium, phonon photon proton subprot sub at at particle point dot line array ray radial dimensionalization/cross time weights with recoherence auto con/etc., hex f, plax, tricho, adherens, coherens, cohesors, adjoiners, junction, junction seal particle point filler retainer, thread strip connector, thread strips, flash image plate, decay, decay return function, flash arc, N-type arc, N type field, imprinting, scale, etc..
DOCTOR DIOBEATLES
BROWN, BROWN BROWN BROWN... BROWNS, ON THE GROUND.
TROY AND PENELOPE
ODYSSEUS AND SIRENS
SCHOFIELD AND TANCRETI (CLASSIC PAIRING)
BUFFY BIFF FREDDY FRED TEDDY
MISTER AND QUEEN CATHERINE AND RUSSIA
ROMANINY AND SUBCRITICAL REACTOR PHENOMENON
WAVY BROWN
TRI LIFIN HANL BAR
BAK IN DE DEY
BACK IN THE DAY...BACK IN NAM...
NAME THE WORM
CANS ANS
AZIEVEETLE & LAYING
CHAUN
CHINGO HONG CHLANG DON
CHIN CHANK
JR Brad Smith
TOM SAWYER & HUCK FINN
GOVENOR & ANNE OF GREEN GABLES
LITTEL HOUSE ON THE PRAIRE
LITTLE WOMEN
MINI WORLD PEOPLE
CHRIS ROODILY ROO ROONEY
AMERICAN TEETLE
LEPPERCHAUN
BLAST TO THE PAST
DROPIN COOLIES OVER THE HIMILAYAS
BRA LEIA
philostigen THEORY
Blas Cross AD and Coolies BC
BLACK BLOOD & CHOLE PALM
COVITTY STAL BLACK STALLION BROOD MARE MINERS MECHANIC MAMA WIFE VIV TIME TIME TIME BLACK BLACK BLACK BLACK BLACK WHITE WHITE WHITE BLACK WHITE WHITE WHITE WHITE BLACK BLACK GREY BLACK BLUE BLACK GREEN HOLOGRAPH DR. F XLIF VIV BIRTHRIGHT SCIENTIST HANDS ARMS CLOAK SUN UNIVERSE DARKS LIGHTS
SCHITZ PSYCHOT DEM GOBLER VEETLE PEATTY AMCAN
CAN AN TING JUZ BLACKY GOLDY
FRECK N BLIND
DINOBEAR
LEP HEP
KRING KRONG
NRILE ROACH KRING OF THE ROACH KRINGDOM
DR LAYNG FANTASY KING
WHINNEY THE SHIT
MENGLE COSTUME
ROUND BROWN
ROMANINOV
RABLES
MIDDLE FEASTY
polish witch, green gang, middle feasty, mengle costume, gosh (gaush), boob beattey, kring krong, goreilla, awhrape, vet, korza, ETC..
CRUM LE CREME
KRINGDOM KRUMB
OUIJI
KLOEN SLABEN
CONSTANTINE
THE WHAYULENT
LEADED APE PELVIS
TRUNK NECK BARREL CHEST BLOWN ROUND HUNCH BACK THUNDER THIGHS LATIN-SKULL LEADED APE PELVIS SAWED OFF HIP JOINTS SUCTION JOINTS OSSIFICANS MINERALIZATION LIQUEFICATION (BIOFILM PLAQUE TARTAR CALCULUS MINERALIZATION CALCIFICATION LIQUEFICATION)
BUSH MATRIX KRIEMER LEISH WIELSHIAN
WEMEL, APE HUMAN HYBRID, FECULAR HUMAN, VERY LOW AMCAN, IRVINE TRAFFICKER, MISTER, ETC., RESEARCH SUBJECTS
GARGOYLE HULK, BANE, CHOLE PALM, BLACK BLOOD, ETC..
SYPHALLI, VASILIKI, AGRANULOCYTE, GLOBULES, PHAG, MAC, PHAGEULENT, AMCAN AGGLOMERATE, CONGLON COGO, CONGO, JUNGLE WOMAN, R TYPE, TARZAN, APE MAN, HUMINAL, WEMEL, ANIMAL HUMAN HYBRID, FECULAR HUMAN, FECES COMP GENERATED HUMAN BIOPHY ORG FORM/LIFEFORM/REPO/SYS/ETC., FERAL CHILD, LEISH, DACF, BLOSK, BRISK, BRINDIL, BETTEY, MAGDEADO, DEDDY FREDDY, FRAN KIN STAYN, SCHITZ, PSYCHOTS, DEMS, PEATTY WADE, WADE LIVER ROCK, ASSPHRODIYTE, DAPHYNE, VELRUMA, SARCLET, SKARLES, SKARLET, SCARLET, PYRE, PETROS, PROX, SARKOTS, JEW, WAVY BRUEN, NEANDER, HEATHEN, CRETIAN, CROTCHIAN, WELSHIAN CHRISTOS, GERMJEW INDO, AMCAN, HOMUNCULUS, HUMUNCULUS, OSSI, FIBRIT, FIBIT, FIRBRIT, BIF, FIBRIT, BI, NEURK, KRIT, KRAT, KRUK, KURK, RUK, BROCK, FROUCH, FRUCH, EADLE, EADEL, FRUT, MOL, HOMO WHROLE MWROEL MRWOLE, ROLE, DTC..
KANDY KANE CANDY CANE
SKRAIPIE OARF PRION PREON FRAG F PILE FRAG PILE
LINE TWIST
SPONGIFORM SPONGE BRAIN GAIT
BRAD
NASTIEST WHITE FEMALE
GEORGE OF THE JUNGLE PSYCHO BLUE IRIS BROWN SPOTTED IRIS WHITE SPOTTING OF THE IRIS
BLOOD SUCKING LEECH
PERDI ET PONGO MARKUS THE DOG KROLE PRALM KWROLE PWRALM RABYS WEB HYPERTROPHIED TRAPEZIUS STEBE SILWVIERS TRUNK NECK
Jaba the hutt
hollow fiber bioreactor, membrane bioreactor
cord rope spine, ferritic core memory
docter diobetteys meningitis encephalitis virus amoeba german trops nagleria plaque demmy cystercercole cytoidial gen scraporf
ortho
The Zoo
Immune system mitigation process, immunosuppression immunodeficiency immune system dysfunction immune disease autoimmune disease reaction sit degeneration immune system failure recalibration leukemia cancot
cancot psychot schitz sarkos/sarkot/SARCLET, cota, mycota, cotic fungyulai, fungulent formules, variety organism proc, development identity, etc..
Sewer people
| Macrophage | |
|---|---|
 A macrophage stretching its "arms" (filopodia)[1] to engulf two particles, possibly pathogens, in a mouse (trypan blue exclusion staining) |
https://en.wikipedia.org/wiki/Macrophage
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