above. Deathstars - Trinity Fields
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Wednesday, September 8, 2021
09-08-2021-1518 - Oxyphilic Adenocarcinoma Follicule oncocytic hurthle cell Thyroid endocrine endocrinology adeno SEER & PUBMED Process "...abundant granular eosinophilic cytoplasm (oncocytes)."
Oxyphilic Adenocarcinoma
- MedGen UID:
- 61427
- •Concept ID:
- C0205642
- •
- Neoplastic Process
Synonyms: | Adenocarcinoma, Oxyphilic; Adenocarcinomas, Oxyphilic; Oxyphilic Adenocarcinomas |
SNOMED CT: | Follicular carcinoma, oxyphilic cell (57596004); Oxyphilic adenocarcinoma (443261008); Oxyphilic adenocarcinoma (57596004); Oncocytic carcinoma (57596004); Oncocytic adenocarcinoma (57596004); Hurthle cell carcinoma (57596004); Hurthle cell adenocarcinoma (57596004) |
09-08-2021-1517 - Myelodysplastic/Myeloproliferative neoplasm, unclassifiable IMMUNOPROLIFERATIVE DISEASES reticular reticulum reticulus endoplasmic reticulum mitochondria cell SEER
NEOPLASMS OF HISTIOCYTES AND ACCESSORY LYMPHOID CELLS IMMUNOPROLIFERATIVE DISEASES
PRECURSOR LYMPHOID NEOPLASMS
LYMPHOID LEUKEMIA, NOS PROLYMPH/PRECURS LEUKEMIA
CHRONIC MYELOPROLIFERATIVE DIS. MYELOPLASTIC/MYELOPROLIFERATIVE NEOPLASMS
975 9759/3 976 9766/3 981 9811/3
Fibroblastic reticular cell tumor Lymphomatoid granulomatosis, grade 3
B lymphoblastic leukemia/lymphoma, NOS
982 9823/3
Chronic lymphocytic leukemia/small lymphocytic lymphoma
9812/3 Leukemia/lymphoma with t(9;22)(q34;q11.2);BCR-ABL1
9813/3 Leukemia/lymphoma with t(v;11q23);MLL rearranged
9814/3 Leukemia/lymphoma with t(12;21)(p13;q22);TEL-AML1(ETV6-RUNX1) 9815/3 B lymphoblastic leukemia/lymphoma with hyperdiploidy
9816/3 Leukemia/lymphoma with hypodiploidy (hypodiploid ALL)
9817/3 B lymphblastic leukemia/lymphoma with t(5;14)(q31;q32);IL3-IGH 9818/3 Leukemia/lymphoma with t(1;19)(q23;p13.3); E2A PBX1 (TCF3 PBX1) 9819/3 B-lymphocytic leukemia/lymphoma, BCR-ABL1-like
T-cell large granular lymphocytic leukemia
983 9831/3
9837/3 T lymphoblastic leukemia/lymphoma
996 9965/3
9967/3 Myeloid and lymphoid neoplasm with FGFR1 abnormalities
Myeloid and lymphoid neoplasms with PDGFRB rearrangement
997 9971/3
9975/3 Myelodysplastic/Myeloproliferative neoplasm, unclassifiable
https://seer.cancer.gov/icd-o-3/sitetype.icdo3.20210607.pdf#search=plasma%20cell
Above. Lil Jon & The East Side Boyz - Get Low
09-08-2021-1515 - SEER Validation List https://seer.cancer.gov/icd-o-3/sitetype.icdo3.20210607.pdf#search=plasma%20cell Muller FBI & Friends
https://seer.cancer.gov/icd-o-3/sitetype.icdo3.20210607.pdf#search=plasma%20cell
(Repost, Original Post Present)
08-24-2021-1053 - Anemia - Autoimmune hemolytic anemia - The term "macrocytic" refers to the enlarged size of the red blood cells.
Autoimmune hemolytic anemia
Anemia (NOS) is a decrease in the number of red blood cells. Many cancer patients develop anemia due to the cancer and/or cancer therapy. This condition may be referred to as Anemia in neoplastic disease or anemia of chronic disorders. The anemia by itself is not reportable. The neoplasm causing the anemia would be reportable.
Aplastic anemia occurs when the bone marrow does not make enough new red cells. A neoplasm may cause the aplastic anemia; however, the aplastic anemia by itself is not reportable. (See also pancytopenia)
Hemolytic (autoimmune) anemia is a group of acute or chronic anemias, inherited or acquired, characterized by shortened survival of mature erythrocytes and inability of bone marrow to compensate for the decreased life span.
Idiopathic refractory anemia is when the cause of the anemia is not known. This is not the same thing as Refractory Anemia(9980/3).
Iron -refractory iron deficiency anemia (IRIDA) or refractory iron deficiency anemia is an autosomal recessive disorder.
Macrocytic anemia is usually caused by vitamin deficiencies, alcohol use, medications or thyroid. The term "macrocytic" refers to the enlarged size of the red blood cells.
Anemia
https://seer.cancer.gov/seertools/hemelymph/532b32a0e4b0626b1926e990/
The term "macrocytic" refers to the enlarged size of the red blood cells.
https://seer.cancer.gov/icd-o-3/sitetype.icdo3.20210607.pdf#search=plasma%20cell
ML, SMALL B-CELL LYMPHOCYTIC
PRECURS. CELL LYMPHOBLASTIC LYMPH.
PLASMA CELL TUMORS
SQUAMOUS CELL CARCINOMA, NOS
GIANT & SPINDLE CELL CARCINOMA
BRONCHIOLO-ALVEOLAR ADENOCA.
CLEAR CELL ADENOCARCINOMA, NOS
ACINAR CELL CARCINOMA
FIBROMATOUS NEOPLASMS
SARCOMA, NOS RHABDOMYOSARCOMA, NOS
EMBRYONAL RHABDOMYOSARCOMA MIXED TUMOR, MALIGNANT, NOS
ML, LARGE B-CELL, DIFFUSE
FOLLIC. & MARGINAL LYMPH, NOS
IMMUNOPROLIFERATIVE DISEASES
LYMPHOID LEUKEMIA, NOS PROLYMPH/PRECURS LEUKEMIA
CHRONIC MYELOPROLIFERATIVE DIS.
OXYPHILIC ADENOCARCINOMA
DUCT CARCINOMA
ACINAR CELL CARCINOMA ADENOSQUAMOUS CARCINOMA
ADENOCA. WITH METAPLASIA
SARCOMA, NOS
FIBROMATOUS NEOPLASMS
SARCOMA, NOS MYXOSARCOMA LIPOSARCOMA NEOPLASMS
MYOMATOUS NEOPLASMS
RHABDOMYOSARCOMA, NOS
MIXED TUMOR, MALIGNANT, NOS CARCINOSARCOMA, NOS
MESENCHYMOMA, MALIGNANT
ML, SMALL B-CELL LYMPHOCYTIC
ML, LARGE B-CELL, DIFFUSE
granuloma sarcoma lymphoma
immunoblastic
FOLLIC. & MARGINAL LYMPH, NOS
NEOPLASMS OF HISTIOCYTES AND ACCESSORY LYMPHOID CELLS
IMMUNOPROLIFERATIVE DISEASES
PRECURSOR LYMPHOID NEOPLASMS
MAL. MEL. IN JUNCT. NEVUS
MAL. MELAN. IN GIANT PIGMT. NEVUS
CLEAR CELL ADENOCARCINOMA, NOS MUCOEPIDERMOID CARCINOMA MUCINOUS ADENOCARCINOMA
PAPILLARY ADENOCARCINOMA, NOS
Lymphomatoid granulomatosis, grade 3
Marginal zone B-cell lymphoma, NOS
8693/3 Extra-adrenal paraganglioma, malignant
MEDIASTINUM C381-C383,C388
THYMUS C379
PLEURA C384
RESPIRATORY,NOS C390,C398-C399
9690/3 Follicular lymphoma, NOS
9691/3 Follicular lymphoma, grade 2
9695/3 Follicular lymphoma, grade 1
9698/3 Follicular lymphoma, grade 3
9699/3 Marginal zone B-cell lymphoma, NOS
BONES & JOINTS (EXCL SKULL AND FACE, MANDIBLE) C400-C403,C408-C409,C412-C414,C418-C419 Dr. Layng Getting Some Brown Right
BLOOD, BONE MARROW, & HEMATOPOIETICSYS C420, C421, C424
FOLLIC. & MARGINAL LYMPH, NOS 969
Acute myelomonocytic leukemia
RETICULO-ENDOTHELIAL C423
Langerhans cell histiocytosis, disseminated
SPLEEN C422
Skin
Peripheral Nerves
Retroperitoneum
(Metastatic proc BK Study USA match)
RETROPERITONEUM & PERITONEUM C480-C482,C488
CONNECTIVE & SOFT TISSUE C490-C496,C498-C499
VAGINA & LABIA C510-C512,C518, C529 (AI or NAI)
CERVIX UTERI C530-C531,C538-C539
CORPUS UTERI C540-C543,C548-C549
UTERUS, NOS C559
OVARY C569
PRECURS. CELL LYMPHOBLASTIC LYMPH.
PLASMA CELL TUMORS
MAST CELL TUMORS
FALLOPIANTUBE C570
OTHER FEMALE GENITAL (EXCL FALLOPIAN TUBE) C571-C574,C577-C579
PLACENTA C589
MULLERIAN MIXED TUMOR
KIDNEY C649
RENAL PELVIS, URETER C659, C669
URINARYBLADDER C670-C679
OTHER URINARYORGANS C680-C681,C688-C689
ORBIT & LACRIMALGLAND, (EXCL. RETINA, EYE, NOS) C690-C691, C693, C695-C698
RETINA C692 - winner nikiya @ nlab
EYEBALL C694
EYE, NOS C699
MENINGES (CEREBRAL,SPINAL) C700-C701,C709
BRAIN, & CRANIAL NERVES, & SPINAL CORD, (EXCL. VENTRICLE, CEREBELLUM) C710-C714, C717-C719, C720-C725
VENTRICLE C715
CEREBELLUM C716
OTHER NERVOUS SYSTEM C728-C729
THYROID GLAND C739
ADRENALGLANDS C740-C741,C749
PARATHYROIDGLAND C750
PITUITARYGLAND C751
CRANIOPHARYNGEALDUCT C752
PINEAL GLAND C753
ICD-0-3 SEER SITE/HISTOLOGY VALIDATION LIST OTHER ENDOCRINE GLANDS C754-C755,C758-C759
ILL-DEFINED C760-C768
LYMPH NODES C770-C775,C778-C779
UNKNOWN C809
N/A 900
https://seer.cancer.gov/icd-o-3/sitetype.icdo3.20210607.pdf#search=plasma%20cell
above. Right Thurr chingy
09-08-2021-1008 - 1. Gas phase (tissue cell expansion by gas collection; nitrogen, voc, protein, virus) 2. fibroti scar inflammation post granulocyte infiltration 3. seppage and holleing with voc agglomerate (dysplasia proliferative granulomatosis cancotics virus gas induction by voc exposure inflam wound heal fibrotics scar deformation gen mod chrom transl/disl/etc. dna cystu w/ gas nuc rad heavy met etc. biofilm plaque tartar calculus mineralization liquefication)
Wednesday, September 8, 2021
09-08-2021-0937 - Primary myelofibrosis (PMF) is a rare bone marrow blood cancer.[1] ' This is most often associated with a somatic mutation in the JAK2, CALR, or MPL gene markers. In PMF, the healthy marrow is replaced by scar tissue (fibrosis), '
Primary myelofibrosis (PMF) is a rare bone marrow blood cancer.[1] It is classified by the World Health Organization (WHO) as a type of myeloproliferative neoplasm, a group of cancers in which there is growth of abnormal cells in the bone marrow. This is most often associated with a somatic mutation in the JAK2, CALR, or MPL gene markers. In PMF, the healthy marrow is replaced by scar tissue (fibrosis), resulting in a lack of production of normal blood cells. Symptoms include anemia, increased infection and an enlarged spleen (splenomegaly).
In 2016, prefibrotic primary myelofibrosis was formally classified as a distinct condition that progresses to overt PMF in many patients, the primary diagnostic difference being the grade of fibrosis.[2]
Primary myelofibrosis | |
---|---|
Other names | PMF, Overt PMF, Myelofibrosis |
Specialty | Oncology and Hematology |
https://en.wikipedia.org/wiki/Primary_myelofibrosis
Arrhythmogenic cardiomyopathy | |
---|---|
Other names | arrhythmogenic right ventricular cardiomyopathy (ARVC), arrhythmogenic right ventricular dysplasia/cardiomyopathy (ARVD/C), right ventricular dysplasia |
Typical micro-histologic features of ARVC/D. Ongoing myocyte death (upper) with early fibrosis and adipocyte infiltration (lower). | |
Specialty | Cardiology |
09-08-2021-0953 - tumor or dysplasia agglomeration or proliferation small capillary tumor/deformity and blood vessel tumor/deformity - NLAB
tumor or dysplasia agglomeration or proliferation
small capillary tumor and blood vessel tumor/deformity
small cell tumor
biofilm tumor
myx xy yxo
Last gen dis, met dis conde, immunosuppr, leukemia/cancer (dysplasia proliferative granulation dysfunction degen etc. matricing widening
cell proliferation metastatic dysplasia adeno
STD CL1 Syphalli, not agranulolytic. granulation disseminative granulation/granulomatoseous (Granulomatosis ) inflammation dna cystulation cystular fibrotics fibromatotics fibrotics sarcotikxs, etc..
Anticipation to CVD,
Granuloma Annulare Lupus erythematosus systemic Lyme Disease Autoimmune Disease
Blood vessel tumor, disseminated vascular lining tumor, shear/frictionated interspace
Arrhythmogenic cardiomyopathy | |
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Other names | arrhythmogenic right ventricular cardiomyopathy (ARVC), arrhythmogenic right ventricular dysplasia/cardiomyopathy (ARVD/C), right ventricular dysplasia |
Typical micro-histologic features of ARVC/D. Ongoing myocyte death (upper) with early fibrosis and adipocyte infiltration (lower). | |
Specialty | Cardiology |
09-08-2021-0949 - Segmental arterial mediolysis (SAM)
Segmental arterial mediolysis (SAM) is a rare disorder of the arteries characterized by the development of aneurysms, blood clots, narrowing of the arteries (stenoses), and blood collections (hematomas) in the affected distribution.[1][2]
SAM most commonly affects the arteries supplying the intestines and abdominal organs.[citation needed]
https://en.wikipedia.org/wiki/Segmental_arterial_mediolysis
Above.
Ricky Martin - Livin' La Vida Loca (Lyrics)
09-08-2021-0948 - Laparotomy for visceral ischemia and gangrene
. 2007 Oct;73(10):1006-8.
Laparotomy for visceral ischemia and gangrene
Karen Woo 1, Kevin Major, Som Kohanzadeh, Alexander D Allins
Affiliations expand
PMID: 17983069
09-08-2021-0947 - Lipofibromatosis-like neural tumor (LPF-NT) lipo fibro matosis fibromatosis fibrosis cancer tumor lipoma
Lipofibromatosis-like neural tumor (LPF-NT) is an extremely rare soft tissue tumor first described by Agaram et al in 2016.[1] As of mid-2021, at least 39 cases of LPF-NT have been reported in the literature.[2][3][4][5] LPF-NT tumors have several features that resemble lipofibromatosis (LPF) tumors,[6] malignant peripheral nerve sheath tumors, spindle cell sarcomas,[3] low-grade neural tumors, peripheral nerve sheath tumors, and other less clearly defined tumors;[1] Prior to the Agaram at al report, LPF-NTs were likely diagnosed as variants or atypical forms of these tumors. The analyses of Agaram at al and subsequent studies[4] uncovered critical differences between LPF-NT and the other tumor forms which suggest that it is a distinct tumor entity differing not only from lipofibromatosis but also the other tumor forms.[7]
LPF-NTs are locally invasive, are commonly treated by surgical excision, and have a relatively high rate of local recurrence if their surgical excisions are incomplete.[8] They are generally considered to be benign, non-metastasizing(i.e. not spreading to other parts of the body) tumors.[6][8] However, one case of LFT-NT reported by Agaram et al was associated with metastasis, apparently as a result of the tumor's cells transformation into a malignant sarcoma. Further studies are needed to determine the frequency of such cases and the overall metastatic potential of LPF-NT.[1]
LPF-NTs were given the "neural tumor" terminology because in at least some cases: 1) their tumor cells express S100 and CD34 but not SOX10 proteins, a pattern that is often found in neural[7] and neuroectodermal tumor cells;[1] and 2) their histopathology consists of tumor cell infiltrations into adipose tissues in a pattern that is very similar to that found in some low grade malignant peripheral nerve sheath tumors.[1]
https://en.wikipedia.org/wiki/Lipofibromatosis-like_neural_tumor
Above. M.I.A. - Paper Planes
09-08-2021-0945 - Fibromuscular dysplasia (FMD)
Fibromuscular dysplasia (FMD) is a non-atherosclerotic, non-inflammatory disease of the blood vessels that causes abnormal growth within the wall of an artery.[1] FMD has been found in nearly every arterial bed in the body although the most common arteries affected are the renal and carotid arteries.[1][2][3]
There are various types of FMD, with multi-focal fibroplasia being the most common. Further, less common, forms of the disease include focal (previously known as intimal) and adventitial fibroplasia.[1][2][3][4] FMD predominantly affects middle-aged women, but has been found in men and people of all ages.[1] Pediatric cases of FMD are vastly different from that of the adult population, and poorly studied. The prevalence of FMD is not known and, although the disease was initially thought to be rare, some studies have suggested that it may be underdiagnosed.[5]
Fibromuscular dysplasia | |
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The "string-of-beads" feature in multi-focal fibromuscular dysplasia. The sign is caused by areas of relative stenoses alternating with small aneurysms. | |
Specialty | Cardiology |
https://en.wikipedia.org/wiki/Fibromuscular_dysplasia
above. Maneater Nelly Furtado
09-08-2021-0942 - Acute myeloid leukemia with mutated RUNX1
Acute myeloid leukemia with mutated RUNX1
Michael Jackson - Billie Jean (Official Video)
09-08-2021-0939 - Extramedullary hematopoiesis (EMH or sometimes EH) hematopoiesis occurring outside of the medulla of the bone (bone marrow).[2] It can be physiologic or pathologic.
Extramedullary hematopoiesis (EMH or sometimes EH[1]) refers to hematopoiesis occurring outside of the medulla of the bone (bone marrow).[2] It can be physiologic or pathologic.
Physiologic EMH occurs during embryonic and fetal development; during this time the main site of fetal hematopoiesis are liver and the spleen.
Pathologic EMH can occur during adulthood when physiologic hematopoiesis can't work properly in the bone marrow and the hematopoietic stem cells (HSC) have to migrate to other tissues in order to continue with the formation of blood cellular components. Pathologic EMH can be caused by myelofibrosis,[3] thalassemias or disorders caused in the hematopoietic system.
Physiologic EMH[edit]
During fetal development, hematopoiesis occurs mainly in the fetal liver and in the spleen followed by localization to the bone marrow.[4] Hematopoiesis also takes place in many other tissues or organs such as the yolk sac, the aorta-gonad mesonephros (AGM) region, the spleen, and lymph nodes. During development, vertebrates go through a primitive and a definitive phase of hematopoiesis. The lungs also play a role in platelet production in adults.[5]
Primitive hematopoiesis[edit]
Primitive hematopoiesis occurs in the yolk sac during early embryonic development. It is characterized by the production of erythroid progenitors or nucleated erythrocytes, also known as erythroblasts or megaloblasts. The main objective of the production of these cells will be the facilitation of tissue oxygenation to support rapid embryonic growth. This primitive phase is transitory and the cells that are produced express embryonic globins, aren't pluripotent, and aren't capable of self-renewal.
Definitive hematopoiesis[edit]
Definitive hematopoiesis differs from the primitive phase through the production of hematopoietic stem cells. The formation of these cells occurs in the AGM later in development. Later, they migrate to the fetal liver where the majority of physiologic EMH takes place. Finally, once the bone marrow has developed, they migrate there. They can also migrate to the spleen and lymph nodes where hematopoiesis can occur, but to a lesser degree.
Pulmonary hematopoiesis[edit]
Pulmonary hematopoiesis also appears to play an important role in adults.[5] In comparison to the bone marrow, where trilineage hematopoiesis occurs, the lungs preferentially contribute to the production of platelets through a resident population of megakaryocytes. This is supported by studies showing that blood leaving the lungs has more platelets and fewer progenitor cells than blood entering the lungs. It has been seen that in cases of severe thrombocytopenia, pulmonary megakaryocytes migrate out of lungs into the bone marrow, where they help to replenish the depleted bone marrow population.
https://en.wikipedia.org/wiki/Extramedullary_hematopoiesis
above.
Ginuwine - Pony
09-08-2021-0938 - Myeloproliferative neoplasms (MPNs)
Myeloproliferative neoplasms (MPNs) are a group of rare blood cancers in which excess red blood cells, white blood cells or platelets are produced in the bone marrow. Myelo refers to the bone marrow, proliferativedescribes the rapid growth of blood cells and neoplasm describes that growth as abnormal and uncontrolled.
The overproduction of blood cells is often associated with a somatic mutation, for example in the JAK2, CALR, TET2, and MPL gene markers.
In rare cases, some MPNs such as primary myelofibrosis may accelerate and turn into acute myeloid leukemia.[1]
Myeloproliferative neoplasm | |
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Other names | Myeloproliferative diseases (MPDs) |
Myelogram of someone with a myeloproliferative disorder. | |
Specialty | Hematology and oncology |
https://en.wikipedia.org/wiki/Myeloproliferative_neoplasm
above.