Genome-wide association studies (GWAS) have identified a number of different loci and genetic pathways that seem to mediate the body's response to allergens and promote the development of allergic rhinitis, with some of the most promising results coming from studies involving single-nucleotide polymorphisms (SNPs) in the interleukin-33 (IL-33) gene.[25][26] T
https://en.wikipedia.org/wiki/Allergic_rhinitis
https://en.wikipedia.org/wiki/Allergic_rhinitis
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Type I/allergy/atopy (IgE) |
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Type II/ADCC |
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Type III (Immune complex) |
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Type IV/cell-mediated (T cells) |
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Unknown/ multiple |
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