https://www.nytimes.com/interactive/2021/us/maine-covid-cases.html
6Million 09122021
NY hospital puts baby deliveries on hold as maternity workers quit over COVID-19 vaccine mandate
Lewis County General Hospital will 'pause' baby deliveries after Sept. 24
Hero purease tank you euthanize missing kids/stolen. Abandon the nurserys my kids/VIVs/birthright/etc. r missing w/ clones-sto ch-disseminated reproductie material/embryo-BTR-Shell swap-orgmiss-amnestic drug DIW indoctrination by USA NAC Amcan et al-etc. (scientist son missing); leave country early w intel val may sanc. dvt gonna up many kids/grown adults need euthan. tank you. USA caused prob petersen-smith-wade-walsh (chris bruen)-layng-aziv-grayden-chaney-silvers-large round yellow betoru-polish witch-romaniov-witch imagers-blbr in med-rooney-wades friends-ocpa-california-amcan-geoffeson-dinurysis-m??xoxo-r??xoxo-latinlawenfoe (bad brown pig, e.g. chris walsh (bad brown pig looses weight = wasting/lean/thin HIV AIDS ST V AND antiviral-medication to effect effemiate and gay-cynical Male Type = IS ID of Chris Walsh Male))-stolen child trafficker fam-irvine trafficker etc.-jewej-whitenig(cauc)-100% caucasian (may start bruen)-norway-latin-MISTER-BB-NATIBETEY-MCL-MCD-Middle easty feasty-Short Layng Friend-Middle East Latin Grayden Accomplices-neander-heathen-germ-barb-cretian-welshianchris-hun (missing hunds)-roman-ispirescu-Wilkes-Wilks-MMM (their missing kids)-Niks missing kids/gene/seq/desc/etc. with bad character, etc.-etc., etc., etc..
Note. Abduction BTR import into USA NAC, Hostage, intel dump cloak amnestic drug clone ships etc. for euthanasia prompt (retrograde all conde unsatisfied by world/human/univ/usa on behalf of usa due usa inferior) bf.=1900s/unk (time ops)[CL-MX to Build, Re-gov to enable proper secret/distanct/etc.], planted CL1 t dr beattey/beatles/beteys/betteys/etc. 1990s (clone shell, etc.), shell swapped out of three or more clone shells or equal shells (brains unk fate; likely reshelled/rebodied post shell swap by USA to accommodate petersen or hybrid brain transplant in to shell that appeared at identity on CL1 to overlap wit agent/dop/birthright/viv/etc. indiscriminate to petersen/petersen followers/usa/etc.; double conts), enhostaged indef dura till re-release intel at CL1. Post-abduction USA enduring inability to follow simple rule and euth (instead dest world by use of anothers tech/etc. and by ip theft).
Birthright ID - euthanasia, blind rights, massive ops, has a pilot, her wars/works unwritten by human (du conflict with their perception and the collapse of reality structure where birthright and viv alone hold; and where they have no need for equal), had a family, missing her shell or not & depending on ape/USA/auth/aggressor/hostage deteiner/etc. demands interim.
Abduct BTR; Improt USA NAC; Hostage; Intel Dump cloak clone amnestic drug f euthan work if correct century detains or more; plant at CL1 in clone/descendant/child/or retained shell at base/etc. as child (not in working adult shell, usually clone may be original descnedent for inbred familiies actual very few in whole world due cloak/USA/Petersen/Smith/ETc.; a true plant singular 92-93 est at NAC CA DOM USA Bettey, eldest daughter nik, was act des/of missing race owned by VIV.unk-DOP, reshelled rebrained), BTR, shell swap, BTR, hostage CL1 name collector and sentences (back val).
https://www.foxnews.com/health/ny-hospital-baby-deliveries-maternity-workers-quit-covid-19-vaccine-mandate
Covid-19 Could Become Like the Flu if More People Get Vaccinated
Virus eventually might behave like a routine illness but getting there will be difficult, as the spread of the Delta variant shows
https://www.wsj.com/articles/covid-19-could-become-like-the-flu-if-more-people-get-vaccinated-11631439002
https://www.nytimes.com/2021/08/25/world/asia/china-coronavirus-covid-conspiracy-theory.html
Round brown (man man; german; fat-shtnk, fibrosis/deformans upn decay; inhaler-ACE2-blood pressure lowering meds-heart meds-etc.) v. bad brown pig (chris walsh; antiviral effemization gay-cynical at Wavy Brown Hair Brown Eye Yellow-Brown-Green/Purple/red/etc. skin tone, 100% caucasian, welsh-bruen, malacia, genetic disease, muscular dystrophy, displasia hyperproliferative inclined granulomateous agglomeration reaction allergy autoimmune incine, gen dis/met dis/resp by autoimmune immunesuppression (genetic lineage/background brain-shell), dental problem conn tiss/mucosa).
- Benzamides
- Cyclobutanes
- Fluoroarenes
- GABAA receptor negative allosteric modulators
- Hormonal antineoplastic drugs
- Imidazolidines
- Lactams
- Nitriles
- Nonsteroidal antiandrogens
- Progonadotropins
- Prostate cancer
- Pyridines
- Spiro compounds
- Thioureas
- Trifluoromethyl compounds
https://en.wikipedia.org/wiki/Antihypertensive_drug
https://en.wikipedia.org/wiki/Apalutamide
https://en.wikipedia.org/wiki/Chlorantraniliprole
https://en.wikipedia.org/wiki/Cobimetinib
https://en.wikipedia.org/wiki/Cariporide
https://en.wikipedia.org/wiki/Category:Benzamides
above. classic trance 1999-2001
above/below. this is my rifle combichrist
Chlorothiazide, sold under the brand name Diuril among others, is an organic compound used as a diuretic and as an antihypertensive.[1][2]
It is used both within the hospital setting or for personal use to manage excess fluid associated with congestive heart failure. Most often taken in pill form, it is usually taken orally once or twice a day. In the ICU setting, chlorothiazide is given to diurese a patient in addition to furosemide (Lasix). Working in a separate mechanism from furosemide and absorbed enterically as a reconstituted suspension administered through a nasogastric tube (NG tube), the two drugs potentiate one another.
It was patented in 1956 and approved for medical use in 1958.[3]
https://en.wikipedia.org/wiki/Chlorothiazide
Thiazide (/ˈθaɪəzaɪd/) refers to both a class of sulfur-containing organic molecules[1] and a class of diuretics based on the chemical structure of benzothiadiazine.[2] The thiazide drug class was discovered and developed at Merck and Co. in the 1950s.[3] The first approved drug of this class, chlorothiazide, was marketed under the trade name Diuril beginning in 1958.[3] In most countries, thiazides are the least expensive antihypertensive drugsavailable.[4]
Thiazide organic molecules are bi-cyclic structures that contain adjacent sulfur and nitrogen atoms on one ring.[5] Confusion sometimes occurs because thiazide-like diureticssuch as indapamide are referred to as thiazides despite not having the thiazide chemical structure.[6] When used this way, "thiazide" refers to a drug which acts at the thiazide receptor.[7] The thiazide receptor is a sodium-chloride transporter that pulls NaCl from the lumen in the distal convoluted tubule. Thiazide diuretics inhibit this receptor, causing the body to release NaCl and water into the lumen, thereby increasing the amount of urine produced each day.[6] An example of a molecule that is chemically a thiazide but not used as a diuretic is methylchloroisothiazolinone, often found as an antimicrobial in cosmetics.[8]
https://en.wikipedia.org/wiki/Thiazide
FATULANT @ CHOLE PALM OD WINC
https://en.wikipedia.org/wiki/Aripiprazole_lauroxil
S-14,506 is a phenylpiperazine, a 5-HT1A receptor agonist, and a dopamine receptorantagonist.
https://en.wikipedia.org/wiki/S-14,506
TC OT 39 is a non-peptide partial agonist of the oxytocin and vasopressin V2receptors (Ki = 147 nM and >1000 nM, respectively) and antagonist of the vasopressin V1A receptor (Ki = 330 nM).[1]
https://en.wikipedia.org/wiki/TC_OT_39
Flecainide is a medication used to prevent and treat abnormally fast heart rates.[1]This includes ventricular and supraventricular tachycardias.[1] Its use is only recommended in those with dangerous arrhythmias or when significant symptoms cannot be managed with other treatments.[1] Its use does not decrease a person's risk of death.[1] It is taken by mouth or injection into a vein.[1][2]
https://en.wikipedia.org/wiki/Flecainide
https://en.wikipedia.org/wiki/Flubendiamide
https://en.wikipedia.org/wiki/IBNtxA
https://en.wikipedia.org/wiki/Category:Phenols
https://en.wikipedia.org/wiki/Category:Drugs_not_assigned_an_ATC_code
https://en.wikipedia.org/wiki/Alatrofloxacin
https://en.wikipedia.org/wiki/2C-B-PP
https://en.wikipedia.org/wiki/Algestone
Acepromazine, acetopromazine, or acetylpromazine (commonly known as ACP, Ace, or by the trade names Atravet or Acezine 2, number depending on mg/ml dose) is a phenothiazine derivative antipsychotic drug. It was used in humans during the 1950s as an antipsychotic,[3] but is now almost exclusively used on animals as a sedative and antiemetic. Its closely related analogue, chlorpromazine, is still used as an antipsychotic in humans. Acepromazine is used primarily as a chemical restraint in hyperactive or fractious animals.
The standard pharmaceutical preparation, acepromazine maleate, is used in veterinary medicine in dogs and cats. It is used widely in horses as a pre-anesthetic sedative and has been shown to reduce anesthesia related death.[4] However, it should be used with caution (but is not absolutely contraindicated) in stallions due to the risk of paraphimosis and persistent priapism.[5] (castratant) Its potential for cardiac effects can be profound, namely hypotension due to peripheral vasodilation, so it should be avoided or used with caution in geriatric or debilitated animals.[6]
Equine[edit]
In equine surgery, premedication with acepromazine has been shown to reduce the perianaesthetic mortality rate, possibly due to its actions as a sedative and anxiolytic.[4] It is less effective as a sedative if the horse is already excited.[23]
Additionally, acepromazine is used as a vasodilator in the treatment of laminitis, where an oral dose equivalent to "mild sedation" is commonly used, although the dose used is highly dependent on the treating veterinarian. While it is shown to elicit vasodilation in the distal limb, evidence showing its efficacy at increasing perfusion in the laminae is lacking. It is also sometimes used to treat a horse experiencing equine exertional rhabdomyolysis.[6]
Adverse effects[edit]
Side effects are not common, but the use of acepromazine in stallions should be used with caution (but is not absolutely contraindicated) due to the risk of paraphimosis and priapism.[5]
Acepromazine also lowers blood pressure, and should therefore be used with caution in horses that are experiencing anemia, dehydration, shock, or colic. It should not be used in horses dewormed with piperazine.[23]
https://en.wikipedia.org/wiki/Acepromazine#cite_note-3
https://en.wikipedia.org/wiki/Priapism
Equine exertional rhabdomyolysis (ER, also known as tying up, azoturia, or Monday-morning disease) is a syndrome that damages the muscle tissue in horses. It is usually due to overfeeding carbohydrates and appears to have a genetic link.
https://en.wikipedia.org/wiki/Equine_exertional_rhabdomyolysis
Both bacterial as well as viral pathogens have been implicated in the etiology of LS. A disease that is similar to LS, acrodermatitis chronica atrophicans is caused by the spirochete Borrelia burgdorferi. Viral involvement of HPV[15] and hepatitis C[16] are also suspected.
A link with Lyme disease is shown by the presence of Borrelia burgdorferi in LSA biopsy tissue.[17]
https://en.wikipedia.org/wiki/Lichen_sclerosus
https://en.wikipedia.org/wiki/8-OH-PBZI
https://en.wikipedia.org/wiki/Allithiamine
https://en.wikipedia.org/wiki/Aliflurane
https://en.wikipedia.org/wiki/Halopropane
https://en.wikipedia.org/wiki/Synthane
https://en.wikipedia.org/wiki/Teflurane
https://en.wikipedia.org/wiki/Category:Benzamides
Benzamide is a white solid with the chemical formula of C6H5C(O)NH2. It is the simplest amide derivative of benzoic acid. It is slightly soluble in water, and soluble in many organic solvents. A number of substituted benzamides are commercial drugs: sulpiride, remoxipride, amisulpride, tiapride, sultopride, veralipride, aminohippuric acid, cisapride, imatinib, and procainamide.
https://en.wikipedia.org/wiki/Benzamide
https://en.wikipedia.org/wiki/ATC_code_N05#N05AL_Benzamides
GRL-0617 is a drug which is one of the first compounds discovered that acts as a selective small-molecule inhibitor of the protease enzyme papain-like protease(PLpro) found in some pathogenic viruses, including the coronavirus SARS-CoV-2. It has been shown to inhibit viral replication in vitro.[1][2][3][4]
https://en.wikipedia.org/wiki/GRL-0617
3CLpro-1 is an antiviral drug related to rupintrivir which acts as a 3CL protease inhibitor and was originally developed for the treatment of human enterovirus 71. It is one of the most potent of a large series of compounds developed as inhibitors of the viral enzyme 3CL protease, with an in vitro IC50 of 200 nM. It also shows activity against coronavirus diseases such as SARS and MERS, and is under investigation as a potential treatment agent for the viral disease COVID-19.[1][2][3][4][5][6][7]
https://en.wikipedia.org/wiki/3CLpro-1
Papain belongs to a family of related proteins with a wide variety of activities, including endopeptidases, aminopeptidases, dipeptidyl peptidases and enzymes with both exo- and endopeptidase activity.[2] Members of the papain family are widespread, found in baculoviruses,[3] eubacteria, yeast, and practically all protozoa, plants and mammals.[2] The proteins are typically lysosomal or secreted, and proteolytic cleavage of the propeptide is required for enzyme activation, although bleomycin hydrolase is cytosolic in fungi and mammals.[4] Papain-like cysteine proteinases are essentially synthesised as inactive proenzymes (zymogens) with N-terminal propeptide regions. The activation process of these enzymes includes the removal of propeptide regions, which serve a variety of functions in vivo and in vitro. The pro-region is required for the proper folding of the newly synthesised enzyme, the inactivation of the peptidase domain and stabilisation of the enzyme against denaturing at neutral to alkaline pH conditions. Amino acid residues within the pro-region mediate their membrane association, and play a role in the transport of the proenzyme to lysosomes. Among the most notable features of propeptides is their ability to inhibit the activity of their cognate enzymes and that certain propeptides exhibit high selectivity for inhibition of the peptidases from which they originate.[5]
https://en.wikipedia.org/wiki/Papain
Sulfur dioxide (IUPAC-recommended spelling) or sulphur dioxide (traditional Commonwealth English) is the chemical compound with the formula SO
2. It is a toxic gas responsible for the smell of burnt matches. It is released naturally by volcanic activity and is produced as a by-product of copper extraction and the burning of sulfur-bearing fossil fuels. Sulfur dioxide has a pungent smell like nitric acid.[citation needed]
https://en.wikipedia.org/wiki/Sulfur_dioxide
Baculoviridae is a family of viruses. Arthropods, lepidoptera, hymenoptera, and dipteraserve as natural hosts. There are 85 known species in this family, assigned to four genera.[1][2][3]
Baculoviruses are known to infect insects, with over 600 host species having been described. Immature (larval) forms of lepidopteran species (moths and butterflies) are the most common hosts, but these viruses have also been found infecting sawflies, and mosquitoes. Although baculoviruses are capable of entering mammalian cells in culture[4]they are not known to be capable of replication in mammalian or other vertebrate animal cells.
Starting in the 1940s they were used and studied widely as biopesticides in crop fields. Baculoviruses contain a circular double-stranded DNA (dsDNA) genome ranging from 80 to 180 kbp.
https://en.wikipedia.org/wiki/Baculoviridae
Itopride (INN) (brand name Ganaton) is a prokinetic benzamide derivative. These drugs inhibit dopamine and acetylcholine esterase enzyme and have a gastrokineticeffect.[3] Itopride is indicated for the treatment of functional dyspepsia and other gastrointestinal conditions.[4] It is a combined D2 receptor antagonist and acetylcholinesterase inhibitor.[5][6]
Itopride is not currently approved by the U.S. Food and Drug Administration (FDA) for use in the United States, nor is it yet approved in the United Kingdom. This may explain the apparent lack of patient information available in English compared to other similar classes of medication.
https://en.wikipedia.org/wiki/Itopride
https://en.wikipedia.org/wiki/Epanolol
Antipsychotics (N05A)
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Antiemetics (A04)
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Drugs for functional gastrointestinal disorders (A03)
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Analgesics (N02A, N02B)
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Authority control Edit this at Wikidata
Categories: Benzamides Phenyl compounds
Benzamide is a white solid with the chemical formula of C6H5C(O)NH2. It is the simplest amide derivative of benzoic acid. It is slightly soluble in water, and soluble in many organic solvents. A number of substituted benzamides are commercial drugs: sulpiride, remoxipride, amisulpride, tiapride, sultopride, veralipride, aminohippuric acid, cisapride, imatinib, and procainamide.
See also[edit]
Aminohippuric acid or para-aminohippuric acid (PAH), a derivative of hippuric acid, is a diagnostic agent useful in medical tests involving the kidney used in the measurement of renal plasma flow. It is an amide derivative of the amino acid glycineand para-aminobenzoic acid that is not naturally found in humans; it needs to be IV infused before diagnostic use.
https://en.wikipedia.org/wiki/Aminohippuric_acid
Diagnostic agents (V04)
Diagnostic agents (V04)
Digestive system
Diabetes
Glucose Tolbutamide
Fat absorption
Vitamin A concentrates
Bile duct patency
Ceruletide Magnesium sulfate Sincalide Sorbitol
Liver functional capacity
Galactose Sulfobromophthalein
Gastric secretion
Betazole Caffeine and sodium benzoate Cation exchange resins Histamine phosphate Methylthioninium chloride Pentagastrin
Exocrine pancreatic function
Bentiromide Pancreozymin cholecystokinin Secretin
Endocrine system
Pituitary function
cortisol Corticorelin Metyrapone GH Pralmorelin Sermorelin Somatorelin
Thyroid function
Protirelin Thyrotropin
Fertility disturbances
Gonadorelin
Tuberculosis
Tuberculin
Renal function
Alsactide Aminohippuric acid Indigo carmine Inulin and other polyfructosans Phenolsulfonphthalein
showvte
Physiology of the kidneys and acid–base physiology
Categories: Amino acid derivativesAnilinesBenzamidesAcetic acids
bart baker aylor Swift - "I Knew You Were Trouble" PARODY
Pathophysiology[edit]
Arsenite impairs nucleotide excision repair,[6] and it may also affect gene expression by increasing or decreasing DNA methylation. The high affinity of arsenic for sulfhydryl groups makes keratin-rich cells (e.g., epidermal keratinocytes) a sensitive target for arsenic-induced toxicity. Arsenic has been shown to alter epidermal keratinocyte differentiation processes,[7] induce overexpression of growth factors,[8] and enhance proliferation of human keratinocytes.
Treatment[edit]
- A chelating agent (e.g., dimercaprol) may be helpful to correct acute arsenic exposure, but it has minimal or no effect for patients who had arsenic exposure a long time ago.
- Oral retinoids (e.g., acitretin,[4][9][10] etretinate[11]) may be helpful in treating arsenic-induced cutaneous lesions and in reducing the risk of cutaneous and internal malignancy formation, especially in Bowman's disease.
- Topical 5-fluorouracil cream[10] or 5% imiquimod cream[12] may be useful in treating arsenical keratoses and Bowen's disease.
Pages in category "Epidermal nevi, neoplasms, and cysts"
The following 200 pages are in this category, out of approximately 261 total. This list may not reflect recent changes (learn more).
(previous page) (next page)A
- Acanthoma fissuratum
- Acanthome cellules claires of Degos and Civatte
- Acquired dermal melanocytosis
- Acrospiroma
- Acrosyringeal nevus of Weedon and Lewis
- Adenoid seborrheic keratosis
- Aggressive digital papillary adenocarcinoma
- Apocrine gland carcinoma
- Apocrine nevus
- Arsenical keratosis
- Atrophic actinic keratosis
- Auricular endochondrial pseudocyst
B
- Balanitis circumscripta plasmacellularis
- Balanitis plasmacellularis
- Balanoposthitis chronica circumscripta plasmacellularis
- Barber's interdigital pilonidal sinus
- Basal cell nevus syndrome
- Basal Cell Nevus syndrome
- Basal cell papilloma
- Basal-cell carcinoma
- Basaloid follicular hamartoma
- Basosquamous cell acanthoma
- Benign lichenoid keratosis
- Birt–Hogg–Dubé syndrome
- Bowenoid papulosis
- Brooke–Fordyce syndrome
- Brooke–Spiegler syndrome
C
- Chondroid syringoma
- Chronic cicatrix keratosis
- Chronic scar keratosis
- Ciliated cyst of the vulva
- Classic basal cell carcinoma
- Clear cell acanthoma
- Clear cell carcinoma of the skin
- Clear cell hidradenoma
- Comedo nevus
- Cornu cutaneum
- Cowden syndrome
- Cowden's disease
- Cutaneous apudoma
- Cutaneous ciliated cyst
- Cutaneous columnar cyst
- Cutaneous horn
- Cylindroma
- Cystic chondromalacia
- Cystic papilloma
D
- Degos acanthoma
- Dermal cylindroma
- Dermal duct tumor
- Dermal eccrine cylindroma
- Dermal melanocyte hamartoma
- Dermatosis papulosa nigra
- Desmoplastic trichoepithelioma
- Digital papillary adenocarcinoma
- Digitate keratoses
- Digitate seborrheic keratosis
- Dilated pore
- Dilated pore of Winer
- Disseminated spiked hyperkeratosis
E
F
- Familial disseminated piliform hyperkeratosis
- Ferguson Smith type of multiple self-healing keratoacanthomas
- Ferguson–Smith syndrome
- Feuerstein and Mims syndrome
- Fibrofolliculoma
- Flegel's disease
- Follicular hybrid cyst
- Follicular-apocrine hamartoma
- Folliculosebaceous cystic hamartoma
- Folliculosebaceous-apocrine hamartoma
G
H
I
L
M
- Malignant acrospiroma
- Malignant chondroid syringoma
- Malignant poroma
- Malignant trichilemmal cyst
- Mantleoma
- Marjolin's ulcer
- Merkel-cell carcinoma
- Microcystic adnexal carcinoma
- Milia en plaque
- Milium (dermatology)
- Minute aggregate keratosis
- Moll's gland cyst
- Morpheaform basal cell carcinoma
- Morphoeic basal cell carcinoma
- Mucinous carcinoma
- Mucinous nevus
- Muir–Torre syndrome
- Multiple familial trichoepithelioma
- Multiple hamartoma syndrome
- Multiple keratoacanthomas of the Ferguson–Smith type
- Multiple minute digitate hyperkeratosis
N
P
- Paget's disease of the breast
- Pale cell acanthoma
- Papillary adenoma
- Papillary eccrine adenoma
- Papillary endothelial hyperplasia
- Papillary hidradenoma
- Papillomatosis cutis carcinoides
- Papillomatosis cutis carcinoides of Gottron–Eisenlohr
- Perifollicular fibroma
- Phakomatosis pigmentokeratotica
- Pigmented actinic keratosis
- Pigmented hairy epidermal nevus syndrome
- Pigmented seborrheic keratosis
- Pilar cyst
- Pilar sheath acanthoma
- Pilar tumor
- Pilonidal sinus
- Pitch keratosis
- Plasma cell balanitis
- Plasma cell vulvitis
- Porcupine man
- Porocarcinoma
- Poroma
- Primary cutaneous adenoid cystic carcinoma
- Primary neuroendocrine carcinoma of the skin
- Primary small cell carcinoma of the skin
- Proliferating epidermoid cyst
- Proliferating epithelial cyst
- Proliferating follicular cystic neoplasm
- Proliferating pilar tumor
- Proliferating trichilemmal cyst
- Proliferating trichilemmal tumor
- Pseudocyst of the auricle
- Pseudoepitheliomatous keratotic and micaceous balanitis
- Pseudoglandular squamous cell carcinoma
- PUVA keratosis
Ichthyosis hystrix, Lambert type[edit]
Also known as ichthyosis hystrix gravior or porcupine man. This disease is characterised by spiny scales which cover the entire body except the face, genitals, palms and soles. The only known cases were in Edward Lambert (known as the porcupine man) who was exhibited in front of the Royal Society in London in 1731 and three generations of his descendants. No cases of this disease are now known though some experts believe that it may have been a type of epidermolytic hyperkeratosis. From the history of the Lambert family the disease appears to have been an autosomal dominant condition.[1][7]
Hystrix-like ichthyosis with deafness syndrome[edit]
HID syndrome is also known as ichthyosis hystrix, Rheydt type after the German city of Rheydt near Düsseldorf where it was first discovered. Symptoms are bilateral hearing loss and spiky hyperkeratotic masses which cover the whole body though the palms and soles are less badly affected. It can be differentiated from KID syndrome which also has symptoms of deafness and ichthyosis by the different distribution of hyperkeratosis. It is an autosomal dominant condition caused by a mutation to the GJB2gene (the same gene affected by KID syndrome).[8][9]
Keratitis–ichthyosis–deafness syndrome (also known as "Ichthyosiform erythroderma, corneal involvement, and deafness," and "KID syndrome,") presents at birth/infancy and is characterized by progressive corneal opacification, either mild generalized hyperkeratosis or discrete erythematous plaques, and neurosensory deafness.[2]:483, 513[3]:565
It is caused by a mutation in connexin 26.[4]
| Keratitis-ichthyosis-deafness syndrome | |
|---|---|
| Other names | "Erythrokeratodermia progressiva Burns"[1] |
https://en.wikipedia.org/wiki/Keratitis–ichthyosis–deafness_syndrome
Senter syndrome is a cutaneous condition characterized by similar skin changes and congenital hearing impairment to keratitis–ichthyosis–deafness syndrome, but is associated with glycogen storage leading to hepatomegaly, hepatic cirrhosis, growth failure and mental retardation.[2]
https://en.wikipedia.org/wiki/Senter_syndrome
Hystrix-like ichthyosis–deafness syndrome (also known as "HID syndrome"[1]) is a cutaneous condition characterized by a keratoderma.[1]
https://en.wikipedia.org/wiki/Hystrix-like_ichthyosis–deafness_syndrome
Acquired[edit]
- Acquired keratodermas
- AIDS-associated keratoderma
- Arsenical keratoses
- Calluses
- Climacteric keratoderma
- Clavi (Corns)
- Eczema
- Human papillomavirus
- Keratoderma blenorrhagicum
- Lichen planus
- Norwegian scabies
- Paraneoplastic keratoderma
- Psoriasis
- Reactive arthritis
- Secondary syphilis
- Tinea pedis
- Sézary syndrome
- Tuberculosis verrucosa cutis
- Drug-induced keratoderma[3]
See also[edit]
- Palmoplantar keratoderma
- Skin lesion
- List of cutaneous conditions
- List of conditions caused by problems with junctional proteins
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